fos (Bioss)
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fos
Fos, supplied by Bioss, used in various techniques. Bioz Stars score: 94/100, based on 2 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+c+fos/c-fos+Polyclonal+Antibody/pmc13092674-21-0-5
Average 94 stars, based on 2 article reviews
Fos, supplied by Bioss, used in various techniques. Bioz Stars score: 94/100, based on 2 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/anti+c+fos/c-fos+Polyclonal+Antibody/pmc13092674-21-0-5
Average 94 stars, based on 2 article reviews
fos - by Bioz Stars,
2026-09
94/100 stars
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Blocking Assay:Article Title: MitoTEMPO modulates trigeminal activation and mitochondrial biogenesis in a nitroglycerin-induced migraine model. Article Snippet: 1 Department of Physiology, Faculty of Medicine, Hacettepe University, Ankara, Turkey 2 Department of Medical Biology, Faculty of Medicine, Hacettepe University, Ankara, Turkey 3 Faculty of Medicine, Department of Physiology SıhhiyeAnkara, Hacettepe University, Ankara 06230, Turkey Abstract Background Migraine is a disabling neurovascular disorder in which neuroinflammation, trigeminal activation, and mitochondrial dysfunction play central roles.. Mitochondria-targeted antioxidants such as mitoTEMPO may modulate these mechanisms; however, their effects in nitroglycerin (NTG)–induced migraine models remain unclear.. This study investigated the effects of mitoTEMPO on mitochondrial biogenesis, fusion–fission dynamics, trigeminal activation, and inflammation in the trigeminal ganglion in an NTG-induced migraine model. Methods and results Thirty male Sprague–Dawley rats were allocated to control, mitoTEMPO (M), NTG, M + NTG (concomitant administration of mitoTEMPO with NTG), and NTG + M (delayed administration of mitoTEMPO following NTG exposure) groups (n = 6/group). Incubation:Article Title: MitoTEMPO modulates trigeminal activation and mitochondrial biogenesis in a nitroglycerin-induced migraine model. Article Snippet: 1 Department of Physiology, Faculty of Medicine, Hacettepe University, Ankara, Turkey 2 Department of Medical Biology, Faculty of Medicine, Hacettepe University, Ankara, Turkey 3 Faculty of Medicine, Department of Physiology SıhhiyeAnkara, Hacettepe University, Ankara 06230, Turkey Abstract Background Migraine is a disabling neurovascular disorder in which neuroinflammation, trigeminal activation, and mitochondrial dysfunction play central roles.. Mitochondria-targeted antioxidants such as mitoTEMPO may modulate these mechanisms; however, their effects in nitroglycerin (NTG)–induced migraine models remain unclear.. This study investigated the effects of mitoTEMPO on mitochondrial biogenesis, fusion–fission dynamics, trigeminal activation, and inflammation in the trigeminal ganglion in an NTG-induced migraine model. Methods and results Thirty male Sprague–Dawley rats were allocated to control, mitoTEMPO (M), NTG, M + NTG (concomitant administration of mitoTEMPO with NTG), and NTG + M (delayed administration of mitoTEMPO following NTG exposure) groups (n = 6/group). |